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Fapstinence: Difference between revisions
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The short-term boost in testosterone in this study, moreover, if replicable, was likely so small and of such a short duration that it would play no practical role in driving differences in behavior, sexual drive, or physiological changes. Hormone studies have discovered it seems to generally require both large,<ref>https://jamanetwork.com/journals/jama/article-abstract/406451</ref> and possibly chronic<ref>https://onlinelibrary.wiley.com/doi/abs/10.1002/ab.10050</ref> doses of testosterone to drive even modest behavioral changes in test subjects. | The short-term boost in testosterone in this study, moreover, if replicable, was likely so small and of such a short duration that it would play no practical role in driving differences in behavior, sexual drive, or physiological changes. Hormone studies have discovered it seems to generally require both large,<ref>https://jamanetwork.com/journals/jama/article-abstract/406451</ref> and possibly chronic<ref>https://onlinelibrary.wiley.com/doi/abs/10.1002/ab.10050</ref> doses of testosterone to drive even modest behavioral changes in test subjects. | ||
Another line no-fappers often point to as evidence of the claim that "no-fap increases T" are rodent studies that find that ejaculation is associated with a short-term reduction in androgen receptor (AR) density in certain regions of the brain. At the same time, sexual abstinence seem to result in the up-regulation of androgen receptor density in the same areas of the rodent brain.<ref>https://pubmed.ncbi.nlm.nih.gov/12914589/</ref> | Another line of research no-fappers often point to as evidence of the claim that "no-fap increases T" are rodent studies that find that ejaculation is associated with a short-term reduction in androgen receptor (AR) density in certain regions of the brain. At the same time, sexual abstinence seem to result in the up-regulation of androgen receptor density in the same areas of the rodent brain.<ref>https://pubmed.ncbi.nlm.nih.gov/12914589/</ref> | ||
Thus, the argument is that an up-regulation of androgen receptor density resulting from abstinence will essentially result in a behaviorally masculinizing effect, as androgen receptors play a decisive role in mediating the effects of androgens on brain function and physiological functions, libido and so on. | Thus, the argument is that an up-regulation of androgen receptor density resulting from abstinence will essentially result in a behaviorally masculinizing effect, as androgen receptors play a decisive role in mediating the effects of androgens on brain function and physiological functions, libido and so on. | ||
It is unlikely that this small increase in androgen receptor density in the brain (assuming this holds true for humans) would play much of a function in altering behavior to any large degree (especially in the absence of large changes in T levels). This neural change likely mainly serves to inhibit excess sexual activity on behalf of male mammals, as the down-regulation in receptor density was the largest among rats that were allowed to copulate ''ad libitum'' with receptive females. | It is unlikely that this small increase in androgen receptor density in the brain (assuming this holds true for humans) would play much of a function in altering behavior to any large degree (especially in the absence of large changes in T levels). This neural change likely mainly serves to inhibit excess sexual activity on behalf of male mammals, as the down-regulation in receptor density was the largest among rats that were allowed to copulate ''ad libitum'' with receptive females. | ||