Fapstinence: Difference between revisions

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The short-term boost in testosterone in this study, moreover, if replicable, was likely so small and of such a short duration that it would play no practical role in driving differences in behavior, sexual drive, or physiological changes. Hormone studies have discovered it seems to generally require both large,<ref>https://jamanetwork.com/journals/jama/article-abstract/406451</ref> and possibly chronic<ref>https://onlinelibrary.wiley.com/doi/abs/10.1002/ab.10050</ref> doses of testosterone to drive even modest behavioral changes in test subjects.
The short-term boost in testosterone in this study, moreover, if replicable, was likely so small and of such a short duration that it would play no practical role in driving differences in behavior, sexual drive, or physiological changes. Hormone studies have discovered it seems to generally require both large,<ref>https://jamanetwork.com/journals/jama/article-abstract/406451</ref> and possibly chronic<ref>https://onlinelibrary.wiley.com/doi/abs/10.1002/ab.10050</ref> doses of testosterone to drive even modest behavioral changes in test subjects.
Another line no-fappers often point to as evidence of the claim that "no-fap increases T" are rodent studies that find that ejaculation is associated with a short-term reduction in androgen receptor (AR) density in certain regions of the brain. At the same time, sexual abstinence seem to result in the up-regulation of androgen receptor density in the same areas of the rodent brain.<ref>https://pubmed.ncbi.nlm.nih.gov/12914589/</ref>
Thus, the argument is that an up-regulation of androgen receptor density resulting from abstinence will essentially result in a behaviorally masculinizing effect, as androgen receptors play a decisive role in mediating the effects of androgens on brain function and physiological functions, libido and so on.
It is unlikely that this small increase in androgen receptor density in the brain (assuming this holds true for humans) would play much of a function in altering behavior to any large degree (especially in the absence of large changes in T levels). This neural change likely mainly serves to inhibit excess sexual activity on behalf of male mammals, as the down-regulation in receptor density was the largest among rats that were allowed to copulate ''ad libitum'' with receptive females.
Thus, these rat studies likely simply prove that no-fap makes one more sexually motivated due to sexual frustation, which is pretty obvious prima facie.


The bulk of scientific research suggests masturbation is not harmful unless it is excessive or occurs in public.<ref>George R. Brown, MD (July 2019). "Overview of Sexuality". Merck Manuals Professional Version. Retrieved 4 March 2020.</ref> Pornography consumption is unlikely to cause inceldom,<ref>https://journals.sagepub.com/doi/full/10.1177/2378023121996854</ref> and even consumption of degenerate porn is unlikely to affect mental functioning as it is generally [[antifragility|hard to perturb]]. Porn consumption is correlated with some negative life outcomes, but this is suspected to be explained by other variables rather than by porn itself.<ref>https://raceandconflicts.home.blog/2021/02/18/on-the-effects-of-pornography/</ref>
The bulk of scientific research suggests masturbation is not harmful unless it is excessive or occurs in public.<ref>George R. Brown, MD (July 2019). "Overview of Sexuality". Merck Manuals Professional Version. Retrieved 4 March 2020.</ref> Pornography consumption is unlikely to cause inceldom,<ref>https://journals.sagepub.com/doi/full/10.1177/2378023121996854</ref> and even consumption of degenerate porn is unlikely to affect mental functioning as it is generally [[antifragility|hard to perturb]]. Porn consumption is correlated with some negative life outcomes, but this is suspected to be explained by other variables rather than by porn itself.<ref>https://raceandconflicts.home.blog/2021/02/18/on-the-effects-of-pornography/</ref>

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