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Looksmaxing: Difference between revisions
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→Hard looksmaxxing
m (→Hard looksmaxxing: moved taking hairloss drugs to hard looksmaxxing, due to the risk of side effects, especially among the more intensive options I added) |
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* Bleaching one's skin or using other chemicals to [[Whitemaxxing|whiten one's skin.]] | * Bleaching one's skin or using other chemicals to [[Whitemaxxing|whiten one's skin.]] | ||
* Various forms of plastic surgery: fillers, laser scar removal, botox etc. | * Various forms of plastic surgery: fillers, laser scar removal, botox etc. | ||
* Preventing male-pattern hair loss with the anti-androgenic five-alpha-reductase enzyme inhibitor drugs finasteride and/or dutasteride. Hair regrowth agents like minoxidil could also be included in this category. In extreme cases, some men experiencing hair loss even opt to take even more potent anti-androgen drugs (drugs that block testosterone production or make it inert in the body) such as spironolactone and abiraterone acetate. This is because testosterone itself is bio-active in the scalp, though to a lesser degree than DHT (a more potent androgen, inhibited by the five-alpha-reductase drugs listed above). So, in some extreme cases, men opt for chemical castration rather than lose their hair. There is also some mechanistic and anecdotal evidence (especially among MTF transsexuals) that estrogen or topically applied estrogen can dramatically stimulate hair regrowth,<ref>https://www.sciencedirect.com/science/article/pii/S0022202X15306242</ref><ref>https://www.pnas.org/content/93/22/12525.short</ref> leading some desperate men experiencing hair loss to experiment with these compounds on their own hair, despite the obvious strong risk of feminization related side effects (gynecomastia, mood swings, disruption of libido, etc). | * Preventing male-pattern hair loss with the anti-androgenic five-alpha-reductase enzyme inhibitor drugs finasteride and/or dutasteride. Hair regrowth agents like minoxidil could also be included in this category. In extreme cases, some men experiencing hair loss even opt to take even more potent anti-androgen drugs (drugs that block testosterone production or make it inert in the body) such as spironolactone and abiraterone acetate. This is because testosterone itself is bio-active in the scalp, though to a lesser degree than DHT (a more potent androgen, inhibited by the five-alpha-reductase drugs listed above). So, in some extreme cases, men opt for chemical castration rather than lose their hair. There is also some mechanistic and anecdotal evidence (especially among MTF transsexuals) that estrogen or topically applied estrogen can dramatically stimulate hair regrowth,<ref>https://www.sciencedirect.com/science/article/pii/S0022202X15306242</ref><ref>https://www.pnas.org/content/93/22/12525.short</ref> leading some desperate men experiencing hair loss to experiment with these compounds on their own hair, despite the obvious strong risk of feminization related side effects (gynecomastia, mood swings, disruption of libido, etc). In the case of topically applied estrogen, the systematic side effects could perhaps be alleviated by the administration of SERMs (selective estrogen receptor modulators), which bind to to estrogen receptor in certain body tissues (such as breast tissue), and are thus often used by steroid using bodybuilders to prevent gynecomastia. | ||
==Controversial looksmaxxing methods== | ==Controversial looksmaxxing methods== | ||